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ویرایش: Second Printing
نویسندگان: Harold D. Foster
سری:
ISBN (شابک) : 1553691326, 9781553691327
ناشر: Trafford Publishing
سال نشر: 2002
تعداد صفحات: 213
زبان: English
فرمت فایل : PDF (درصورت درخواست کاربر به PDF، EPUB یا AZW3 تبدیل می شود)
حجم فایل: 749 کیلوبایت
کلمات کلیدی مربوط به کتاب چه چیزی واقعا باعث ایدز می شود: بیماری ایدز بیماری های جسمی سلامت تناسب اندام رژیم غذایی HIV سرطان مغز قلبی عروقی دیابت قابل انتقال اندام های گوارشی اندام ها پسوریازیس آسیب شناسی ویروسی طب داخلی
در صورت تبدیل فایل کتاب What Really Causes AIDS به فرمت های PDF، EPUB، AZW3، MOBI و یا DJVU می توانید به پشتیبان اطلاع دهید تا فایل مورد نظر را تبدیل نمایند.
توجه داشته باشید کتاب چه چیزی واقعا باعث ایدز می شود نسخه زبان اصلی می باشد و کتاب ترجمه شده به فارسی نمی باشد. وبسایت اینترنشنال لایبرری ارائه دهنده کتاب های زبان اصلی می باشد و هیچ گونه کتاب ترجمه شده یا نوشته شده به فارسی را ارائه نمی دهد.
WHAT REALLY CAUSES AIDS:
AN EXECUTIVE SUMMARY The AIDS pandemic is likely to become the
greatest catastrophe in human history. Unless a safe, effective
vaccine is quickly developed, or the preventive strategies
outlined in this book are widely applied, by 2015 one sixth of
the world's population will be infected by HIV-1 and some 250
million people will have died from AIDS. Its associated losses
by then will be more than those of the Black Death and World
War II combined, the equivalent of eight World War Is.1 This
pandemic is only one of several ongoing catastrophes involving
viruses that encode the selenoenzyme glutathione peroxidase.2
Indeed, the world is experiencing simultaneous pandemics caused
by Hepatitis B and C viruses, Coxsackie B virus and HIV-1 and
HIV-2. As these viruses replicate, because their genetic codes
include a gene that is virtually identical to that of the human
enzyme glutathione peroxidase, they rob their hosts of
selenium. Paradoxically, however, they diffuse most easily in
populations that are very selenium deficient,3 possibly because
their members have depressed immune systems. It is no
coincidence that such viruses are causing havoc at the
beginning of the 21st century. The last 50 years have seen
enormous expansions in the use of fossil fuels and
deforestation by fire. The resulting pollutants have greatly
increased the acidity of global precipitation, reducing
selenium's ability to enter the food chain. This situation is
being made worse by the widespread use of commercial
fertilizers since their sulphates, nitrogen, and phosphorus all
depress the uptake of selenium by crops. Deficiencies in this
essential trace element are being felt most acutely in areas,
such as sub-Saharan Africa, where soil selenium levels are
naturally very low. Acid rain is making a bad situation worse,
so increasing vulnerability to those viruses that encode
glutathione peroxidase. Many populations are also being exposed
to a thinning ozone layer, heavy metals such as mercury and
cadmium, pesticides, and drug, tobacco, and alcohol abuse, all
of which depress the human immune system, increasing
vulnerability to viruses, including HIV-1 and HIV-2. In July
2000, physicians and scientists from around the world met in
Durban, South Africa for the XIII International AIDS
Conference. In a declaration, named after the city, 5,018 of
them proclaimed that "HIV is the sole cause of AIDS."4 There
are, however, at least seven anomalies that strongly suggest
that this conventional wisdom is incorrect and that belief in
it is blocking progress in the development of new treatments
for AIDS and of novel ways of preventing its spread. To
illustrate, despite widespread unprotected promiscuous sexual
activity in Senegal, HIV- 1 is diffusing very slowly, if at
all, amongst the Senegalese.5 It is very apparent that in
Africa, differences in soil selenium levels are greatly
influencing who becomes infected with HIV-1 and who does not.
Indeed, the recently published Selenium World Atlas used the
incidence of HIV-1 as a surrogate measure of soil selenium
levels because actual levels are, as yet, poorly established in
sub-Saharan Africa. A similar relationship has been documented
in the United States6 where there has been an inverse
relationship, especially in the Black population, between
mortality from AIDS and local soil selenium levels. It is well
established that individuals who are HIV-positive gradually
become more and more selenium deficient.7 This decline, which
is known to undermine immune functions, is not unique to
HIV-infection but is seen in almost all infectious pathogens.8
However, under normal circumstances, where death does not
occur, selenium levels rebound soon after recovery. HIV-1,
however, can effectively elude the defense mechanisms of the
immune system, and can continue to replicate indefinitely,
endlessly depressing serum selenium. As a result, the immune
system is compromised, allowing infection by other pathogens
that continue to deplete the host of selenium, allowing HIV-1
to replicate more easily, further undermining immunity.
Therefore, this relationship between selenium and the immune
system is one of positive feedback, in which a decline in
either of these two variables causes further depression in the
other. Termed the "selenium- CD4 T cell tailspin" by the
author,9 it is the reason that serum selenium levels are a
better predictor of AIDS mortality than CD4 T cell counts. Like
other positive feedback systems, such as avalanches and forest
fires, it is extremely difficult to control and gains momentum
as it progresses. HIV-1, however, encodes the entire
selenoenzyme, glutathione peroxidase. As it replicates,
therefore, it depletes its host not only of selenium but also
of the other three components of this enzyme: namely, cysteine,
glutamine, and tryptophan.10 AIDS, therefore, is a nutritional
deficiency illness caused by a virus. Its victims suffer from
extreme deficiencies of all four of these nutrients which are
responsible for such symptoms as depressed CD4T lymphocyte
count, vulnerability to cancers (including Kaposi's sarcoma),
depression, psoriasis, diarrhea, muscle wasting, and dementia.
Associated infections cause their own unique symptoms and
increased risk of death. HIV-1 alone, therefore, does not cause
AIDS. It involves a multiplicity of co-factors, specifically
anything that either depletes serum selenium levels or
depresses the immune system enough to permit viral replication.
Manipulating the "selenium-CD4T cell tailspin" by adding this
trace element to fertilizers and food stuffs opens new avenues
for both prevention and treatment. This strategy has been shown
to work on other viruses that encode glutathione peroxidase,
such as Hepatitis B and C and the Coxsackievirus. The logical
treatment of AIDS patients involves supplementation with
selenium, cysteine, glutamine, and tryptophan, at least to
levels at which deficiency symptoms associated with a lack of
these nutrients disappear. While this can be most easily
achieved by supplements, certain foods contain elevated levels
of those four nutrients. Strangely enough, one of the ideal
meals for anyone who is HIV-seropositive would include a
cheeseburger to which Brazilnut flour had been added to the
bun. REFERENCES
1. Foster, H.D. (1976). Assessing disaster magnitude: A social
science approach. The Professional Geographer, xxviii(3),
241-247.
2. Taylor, E.W. (1997). Selenium and viral diseases: Facts and
hypotheses. Journal of Orthomolecular Medicine, 12 (4),
227-239.
3. Ibid.
4. The Durban Declaration (2000). Nature, 406, 15-16.
5. UNAIDS/WHO Epidemiological Fact Sheet on HIV/AIDS and
sexually transmitted infections: Senegal. 2000 update
(revised).
6. Cowgill, U.M. (1997). The distribution of selenium and
mortality owing to acquired immune deficiency syndrome in the
continental United States. Biological Trace Element Research,
56, 43-61.
7. Baum, M.K., Shor-Posner, G., Lai, S., Zhang, G., Lai, H.,
Fletcher, M.A., Sauberlich, H., and Page, J.B. (1997). High
risk of HIV-related mortality is associated with selenium
deficiency. Journal of Acquired Immune Deficiency Syndromes and
Human Retrovirology, 15(5), 370- 374.
8. Sammalkorpi, K., Valtonen, V., Alfthan, G., Aro, A., and
Huttunen, J. (1988). Serum selenium in acute infections.
Infection, 16(4), 222- 224.
9. Foster, H.D. (2000). Aids and the "selenium-CD4T cell
tailspin": The geography of a pandemic. Townsend Letter for
Doctors and Patients, 209, 94-99.
10. Mariorino, M., Aumann, K.D., Brigelius-Flohe;, R., and
Doria, D., van den Heuvel, J., McCarthy, J.E.G., Roveri, A.,
Ursini, F., and Flohé, L. (1998). Probing the presumed
catalytic triad of a seleniumcontaining peroxidase by
mutational analysis. Z. Ernahrungswiss, 37(Supplement 1),
118-121.