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ویرایش: 4 نویسندگان: Mark J. Ashley, David A. Hovda - Traumatic Brain Injury_ Rehabilitation, Treatment, and Case Management, Fourth Edition-CRC Press (2018).pdf سری: ISBN (شابک) : 9781498710299 ناشر: CRC press سال نشر: 2018 تعداد صفحات: 1297 زبان: English فرمت فایل : PDF (درصورت درخواست کاربر به PDF، EPUB یا AZW3 تبدیل می شود) حجم فایل: 12 مگابایت
در صورت تبدیل فایل کتاب Traumatic Brain Injury_ Rehabilitation, Treatment, and Case Management, Fourth Edition-CRC Press (2018) به فرمت های PDF، EPUB، AZW3، MOBI و یا DJVU می توانید به پشتیبان اطلاع دهید تا فایل مورد نظر را تبدیل نمایند.
توجه داشته باشید کتاب آسیب مغزی تروماتیک - توانبخشی، درمان و مدیریت مورد، چاپ چهارم-CRC (2018) نسخه زبان اصلی می باشد و کتاب ترجمه شده به فارسی نمی باشد. وبسایت اینترنشنال لایبرری ارائه دهنده کتاب های زبان اصلی می باشد و هیچ گونه کتاب ترجمه شده یا نوشته شده به فارسی را ارائه نمی دهد.
Cover Half Title Title Page Copyright Page Table of Contents Preface Acknowledgments Editors Contributors Section 1: Neuroscience Chapter 1: Bioscience indications for chronic disease management and neuromedical interventions following traumatic brain injury Introduction Loss of Axonal Integrity After TBI Blood–Brain Barrier Microglia Cerebral inflammation is mediated by microglia Microglial alterations following TBI Inflammation and axonal damage The dual action of microglia and cytokines Mitochondria Myelination Neuroendocrine function Somatotrophic axis Gonadotroph axis Testosterone Estrogen Progesterone Thyrotroph axis Clinical implications and potential therapeutics References Chapter 2: The neurobiology of traumatic brain injury Introduction What happens to the cells in the brain following TBI? Outcome measurements in experimental animal models Long-term issues with TBI Experimental animal models of CTE as defined by phosphorylated tau Summary and conclusions References Chapter 3: Repeat traumatic brain injury models Introduction The models Experimental design Repeat mild TBI in developing animal models Adolescent RTBI Histology and behavior Brain impact interval influences metabolism and amyloid deposition Pituitary dysfunction Gender Chronic pathology Prepubertal repeat TBI Characterization of pathology time course Repeat mild TBI in adult animal models Metabolism Inflammation Axonal injury Acute and chronic behavioral profiles Neurodegenerative diseases Summary References Chapter 4: Neuroplasticity and rehabilitation therapy Introduction Habituation Sensitization Types of learning Hierarchical learning Multimodal rehabilitation Neurogenesis in adult humans Constraint-induced therapy Summary References Chapter 5: Environmental enrichment: A preclinical model of neurorehabilitation for traumatic brain injury Traumatic brain injury is a significant health care issue EE defined EE as a potential therapeutic approach: early support EE as a potential therapeutic approach: further support Benefits of EE are not gender dependent Benefits of EE are enduring Therapeutic window of EE is flexible EE: combination therapy paradigms Potential mechanisms mediating EE-induced benefits Caveats to the EE effect Conclusion Acknowledgments References Chapter 6: Neuroanatomy of basic cognitive function Introduction Sensory systems Reticular Formation Hypothalamus, pituitary, thalamus, and basal ganglia Hypothalamus and Pituitary Thalamus Basal Ganglia Medial Temporal Lobe and Hippocampal Complex Inferior Temporal Lobe Frontal Lobe Commissural and Association Tract Fibers Principles of Neurophysiology and Cognition Information Processing, Neurotransmission, and Learning Neuromodulatory neurotransmitters Networks and Cognitive Function Specific functions Working Memory Decision-Making Perceptual Decision-Making Prospective Memory Default mode network DMN: Hubs and subsytems DMN functional significance Frontoparietal attention networks Summary References Chapter 7: TBI rehabilitation: Lessons learned from animal studies about mechanisms, timing, and combinatorial approaches Introduction Rehabilitation promotes recovery of function and plasticity Rehabilitation and plasticity following TBI Rehabilitation Timing Combining rehabilitation with adjunctive therapies: What’s the winning combination? Conclusions and future directions References Chapter 8: Diet and exercise interventions to promote metabolic homeostasis in TBI pathology Introduction Lifestyle and Mental Health The Metabolic Pathology of TBI BDNF: A Link Between Metabolic and Cognitive Dysfunctions in the Pathology of TBI Role of Nutritional Factors in Normal Brain Health And After Tbi Omega-3 fatty acids Plasma membrane is susceptible to the effects of TBI and diet The Antioxidant Action of Vitamin E on TBI Dietary Polyphenols and Cognitive Performance: Curcuminoids Dietary Polyphenols and Cognitive Performance: Resveratrol Dietary Flavonoids and Cognitive Function Metabolic disturbances as a signature of TBI pathology Diet and Epigenetics: A Platform for Extending Action and Influencing Neurological Disorders Effect of Exercise on Brain Health and Repair Collaborative Effects of Diet and Exercise Conclusion References Chapter 9: Disruptions in physical substrates of vision following traumatic brain injury Introduction Vision General anatomy Core components Anatomic components Cortical organization Neocortical modules Neural networks Anatomical divisions of visual system Image system Optical system Primary visual system Optic radiation Secondary visual system Nonimage system Retinohypothalamic tract Principles of visual system organization Functional component of the visual system Reception Primary Visual System Oculomotor system Attention Arousal/alerting Orienting network Selective attention network Higher visual processing Ventral stream Dorsal stream Beyond the ventral/dorsal stream TBI Definition Primary effect Secondary effects Classifications of visual deficit Receptive system Attention system Higher visual processing system Summary References Chapter 10: Potential utility of resting state fMRI–determined functional connectivity to guide neurorehabilitation Introduction Determination of functional connectivity with rsfMRI Methods used to determine network FC Major brain networks Potential physiologic correlates of altered FC Current evidence for FC as an outcome measure for injury and rehabilitation Functionally guided rehabilitation—The future? References Chapter 11: TBI and sensory sensitivity: Translational opportunities Introduction Visual Sensitivity Auditory Sensitivity Experimental Approaches Treatments Conclusion References Chapter 12: The neuroimaging challenges in hemispherectomy patients Introduction History Associated Complications Neuroimaging Hemispherectomy Patients Data processing challenges Results Discussion Conclusion References Section 2: Medical Chapter 13: Clinical management of the minimally conscious state Introduction Disorders of Consciousness: Overview and Definitions Diagnosis of MCS Clinical expertise Standardized behavioral assessment CRS-R SSAM WHIM WNSSP SMART DOCS Individualized quantitative behavioral assessment Advanced neuroimaging and neurophysiology (PET, fMRI, EEG) Clinical interventions Avoiding complications Sensory stimulation/regulation Neuromodulation Conclusion Acknowledgments References Chapter 14: Neuropharmacologic considerations in the treatment of vegetative state and minimally conscious state following brain injury Introduction Definitions of coma, vegetative state, minimally conscious state, and functional locked-in syndrome Neurophysiology of arousal and consciousness Functional neuroimaging in disorders of consciousness Prognosis of disorders of consciousness Enhancing the potential for recovery from vegetative state and MCS Establish a baseline of neurologic function Rule out treatable causes of failure to improve Intracranial Complications Endocrine Dysfunction Subclinical Seizure Activity Laboratory Testing Malnutrition Sleep Disturbance Eliminate or reduce sedating medications Pharmacologic intervention to enhance arousal and responsiveness Catecholaminergic neuromodulation Dopaminergic neuromodulation Noradrenergic neuromodulation Methylphenidate Amphetamines Atomoxetine Tricyclic Antidepressants GABA neuromodulation Zolpidem (Ambien) Benzodiazepines Glutamatergic neuromodulation Modafinil Cholinergic neuromodulation Histaminergic neuromodulation Conclusion References Chapter 15: Clinical management of pituitary dysfunction after traumatic brain injury Pathophysiology of TBI-induced pituitary dysfunction Pediatric TBI Sports-related TBI Clinical symptoms of hypopituitarism Guidelines for screening for hypopituitarism Endocrine testing Replacement therapy rationale Summary References Chapter 16: Neurotransmitters and pharmacology Editor’s note Introduction Chemical neurotransmission Sites where drugs act Acetylcholine (ACh) Synthesis, storage, release, and inactivation of ACh Acetylcholine receptors Nicotinic receptors Muscarinic receptors Clinically useful drugs that alter cholinergic neurotransmission Facilitators of cholinergic neurotransmission Inhibitors of cholinergic neurotransmission Cholinergic drugs in the TBI patient Norepinephrine Synthesis, storage, release, and inactivation of NE Norepinephrine receptors Clinically useful drugs that alter noradrenergic neurotransmission Facilitators of noradrenergic neurotransmission Inhibitors of noradrenergic neurotransmission Noradrenergic drugs in the TBI patient Dopamine Synthesis, storage, release, and inactivation of dopamine Dopamine receptors Clinically useful drugs that alter dopamine neurotransmission Facilitators of dopaminergic neurotransmission Inhibitors of dopaminergic neurotransmission Dopaminergic drugs in the TBI patient 5-Hydroxytryptamine (serotonin) Synthesis, storage, release, and inactivation of serotonin Serotonin receptors Clinically useful drugs that alter serotonergic neurotransmission Facilitators of serotonergic neurotransmission Inhibitors of serotonergic neurotransmission Serotonergic drugs in the TBI patient Gamma aminobutyric acid (GABA) Synthesis, storage, release, and inactivation of GABA GABA receptors Clinically useful drugs that alter GABAergic neurotransmission Facilitators of gabaergic neurotransmission Inhibitors of gabaergic neurotransmission GABAergic drugs in the TBI patient Glycine Synthesis, storage, release, and inactivation of glycine Glycine receptors Clinically useful drugs that alter glycinergic neurotransmission Glycinergic drugs in the TBI patient L-glutamic acid Synthesis, storage, release, and inactivation of glutamate Excitatory amino acid neurotransmitter receptors Clinically useful drugs that alter excitant amino acid neurotransmission Drugs that enhance the action of glutamate Drugs that inhibit the action of glutamate Glutamatergic drugs in the TBI patient Peptide neurotransmitters Opioid peptides as neurotransmitters Synthesis, storage, release, and inactivation of opioid peptides Opioid receptors Clinically useful drugs that alter opioid neurotransmission Drugs that enhance opioidergic neurotransmission Drugs that inhibit opioidergic neurotransmission Opioids in the TBI patient Summary References Chapter 17: Pituitary dysfunction after traumatic brain injury Introduction Prevalance of TBI-induced hypopituitarism Adults Children Natural history Sports Pathophysiology of TBI-induced hypopituitarism Anatomy and location of the pituitary Inflammation and autoimmunity Hormones axes disrupted by TBI TBI-induced anterior pituitary dysfunction Growth hormone Gonadal Adrenal Thyroid Prolactin TBI-induced posterior pituitary dysfunction Antidiuretic hormone Clinical manifestation of TBI-induced hypopituitarism Impairments in neurocognition Neuropsychiatric disability and QoL Physical appearance and sexual health Adolescent development TBI-induced hypopituitarism: How to test? Patient screening for pituitary dysfunction Timing of testing for pituitary dysfunction TBI-induced hypopituitarism: When to treat? TBI-induced hypopituitarism: Current basic research Summary References Chapter 18: Increasing physiologic readiness to improve functional independence following neurotrauma Introduction Basic foundations Sleep Nutrition Analgesia Hydration Exercise Pharmacological enhancement strategies Cognitive stabilization Anergia intervention Caveat: Dyscompliance versus noncompliance after frontal lobe injury Conclusion References Chapter 19: Assessment and management of mild traumatic brain injury Introduction Definition of MTBI/concussion Heterogeneity of injury Prevalence Symptoms and dysfunction following MTBI/concussion Postconcussion syndrome Diagnosis and documentation Treatment History in the making References Chapter 20: Chronic traumatic encephalopathy Introduction Neuropathology History of the neuropathological characterization Gross pathology of CTE Hyperphosphorylated tau pathology in CTE Staging of p-tau pathology Axonal pathology in CTE TDP-43 pathology in CTE Aß pathology in CTE CTE as a comorbidity in neurodegenerative disease brain banks CTE with ALS Pathological distinctions between CTE and AD or aging Clinical syndrome Clinical diagnosis of CTE Risk and protective factors Is CTE caused by trauma? Biomechanisms of CTE neurodegeneration Tau propagation in the CNS Other pathogenetic considerations Summary References Chapter 21: Posttraumatic epilepsy and neurorehabilitation Introduction Evaluation of episodic behavioral changes Clinical evaluation of seizures Etiologic considerations Diagnostic investigations of posttraumatic seizures Potential epileptogenesis associated with psychotropic medications Therapy for posttraumatic epilepsy Mechanisms and models of posttraumatic epilepsy Posttraumatic seizures, epilepsy, and anticonvulsant prophylaxis: Implications for neurobehavioral recovery Conclusions Acknowledgments References Section 3: Therapy Chapter 22: Evaluation of traumatic brain injury following acute rehabilitation Introduction Preparation Evaluation Current medical status Audiometry Cognition Education Family Occupational/physical therapy Psychosocial Speech/language pathology Vision Productive activity/vocation Report preparation Summary References Appendix 22-A: Patient examination report Template Appendix 22-B: Iconic store cards Appendix 22-C: Oral peripheral evaluation Form Chapter 23: Neuropsychology following brain injury: A pragmatic approach to outcomes, treatment, and applications Introduction Neuropsychological outcomes and treatment of brain injury Neurocognitive sequelae and outcomes of brain injury Confounding factors affecting outcomes Psychological Factors Pain Sleep ALCOHOL AND Substance Abuse Computerized versus traditional testing Treatment Traditional Rehabilitation Training Computerized CR Training Comparison of Traditional and Computerized Rehab Training Neuropsychological referrals When to make the referral Deconstructing the report Neuropsychological recommendations Suggestions on providing feedback to the patient Information for caregivers Conclusion References Chapter 24: Neuropsychological interventions following traumatic brain injury Introduction Neuropsychological rehabilitation Principles of neuropsychological rehabilitation From isolation to integration: The evolution of comprehensive treatment Is there evidence that cognitive rehabilitation works? Recurrent themes in neuropsychological rehabilitation The apparent dichotomy between restorative and compensatory interventions The selection of appropriate outcome measures The timing of the intervention Neuropsychological interventions: Some highlights Interventions for cognition Attention Memory Executive Function Interventions for emotion Interventions for self-awareness Future directions in neuropsychological rehabilitation Technology and neuropsychological rehabilitation Conclusion Acknowledgments References Chapter 25: The use of applied behavior analysis in traumatic brain injury rehabilitation Introduction The brain–behavior relationship Medication Ethics General management guidelines Basic principles Antecedent Behavior Consequence Prompting and fading Generalization Behavioral diagnostics Historical survey Current status Functional assessment Behavior plan format Goals Target behavior Rationale Materials and data collection Treatment procedures Contraindications Behavior plan procedures Accelerative programs Positive programming Shaping Chaining Decelerative programs Differential reinforcement of incompatible behaviors (dri) Differential reinforcement of other behaviors (dro) Differential reinforcement of low rates of behavior (drl) Overcorrection Stimulus change Stimulus satiation Time out Complex programs Contracting Stimulus control Token economies Other programs Noncontingent reinforcement (ncr) Summary Data collection and graphing Data collection Event recording Interval recording Time sample recording Data management Graphing Crisis prevention and intervention Models of assault Identification model Response models General techniques and methods Staff and family training Putting it all together Concluding remarks References Chapter 26: Rehabilitation and management of visual dysfunction following traumatic brain injury Introduction Physical substrates of vision Multidisciplinary approach Prevalence and impact of visual dysfunction in TBI patients Therapeutic intervention: What and why? Plasticity and flexibility in the adult visual system Remediation of ocular–motor and binocular disorders following TBI Management of other visual dysfunctions following TBI When to treat A useful model for organizing visual rehabilitation Sensory input/reception Perception/integration/attention Motor output/behavior Visual thinking/memory (visual cognition) Assessment and rehabilitation of the visual system Assessment and rehabilitation of sensory input/reception Eye movements Binocular dysfunction Decreased visual acuity Decreased contrast sensitivity Visual field loss Blindsight Photophobia Intolerance of busy spaces Assessment and rehabilitation of perception/integration/attention Localization and spatial vision Visual–spatial neglect Object perception Assessment and rehabilitation of motor output/behavior Eyes Hands Body Assessment and rehabilitation of visual thinking/memory (visual cognition) Summary Illustrative visual case studies Patient J. G. Patient J. R. Patient C. L. Patient L. R. Patient B. B. Acknowledgments References Appendix 26-A Chapter 27: Remediative approaches for cognitive disorders after TBI Introduction Cognition Attention Perceptual features Categorization Cognitive distance Assessment Tools Conditions for Cognitive Rehabilitation Therapeutic intervention Attention Memory Feature identification Categorization Neuroanatomy of the cognition module Summary References Chapter 28: Principles of cognitive rehabilitation in TBI: An integrative neuroscience approach Introduction Models of cognitive rehabilitation Cognitive theory and rehabilitation Effects of Brain Injury on Neuronal Function Cell function/cell death Diffuse axonal injury (DAI) Metabolic dysfunction Brain reorganization and sprouting following injury General Principles of Cognitive Systems Domains of cognition Attention Memory Systems and Processes Verbal Language Categorization Executive Functioning Neuroanatomical correlates of EF EF domains Directions for rehabilitation Efficacy Research Conclusions References Appendix 28-A Part A Level 1: Perceptual feature identification and application Level 2: Similarities and Differences Level 3: Functional Categorization Level 4: Analogies Level 5: Abstract Word Categorization Part B Level 1: Progressive Rule Learning 1 Level 2: Progressive Rule Learning 2 Level 3: Progressive Rule Learning 3 CP-related dependent measures Chapter 29: Management of residual physical deficits Introduction Evaluative process Range of motion, flexibility, and dexterity Neurological examination Sensation and proprioception Deep tendon reflexes and pathological reflexes Cerebellar tests Rapid, alternating movement evaluation Manual muscle test Muscle tone Muscle and cardiovascular endurance Mobility, posture, and gait evaluations Vestibular evaluation Sensorimotor integration and dynamic balance evaluation Assessment of smell and taste Evaluation of vision Visual perception and perceptual motor evaluation Assessment of activities of daily living Concomitant injuries Orthopedic and Spinal Cord TMJ Dysfunction Pain Driving Functioning at heights Management of residual physical deficits Therapeutic measurement Mobility Abnormal tone/spasticity Pain Postural control and balance Cerebellar dysfunction Sensory function Hemiparetic limb and CIMT Smell and taste Visual perception and perceptual motor functions Driving Cardiovascular fitness Leisure Pool/aquatic therapy Summary References Chapter 30: Undertaking vocational rehabilitation in TBI rehabilitation Introduction Employment trends following TBI Prognosticating RTW Industry-related factors influencing RTW VR Prerequisites Using the ICF model to inform RTW planning and interventions Injury-related factors influencing RTW Physical deficits Psychological and behavioral issues Cognitive deficits Communicative deficits RTW models Formalized VR in TBI Prevocational counseling Vocational evaluation VR plan development Vocational Counseling Vocational testing/work evaluation/work hardening Follow-up Summary References Section 4: Case management Chapter 31: Contribution of the neuropsychological evaluation to traumatic brain injury rehabilitation Introduction Historical context of neuropsychology Origins of the term “neuropsychology” Neuropsychological evaluation process Neuropsychological testing versus neuropsychological evaluation Components of the neuropsychological evaluation Neuropsychological evaluation approaches Fixed-Battery Approach Flexible-Battery Approaches Content of the neuropsychological evaluation Cognitive screening and mental status examinations Ecological Implications Global level of performance Ecological Implications Sensory–motor integrity Ecological Implications Language functioning and pragmatics of communication Ecological Implications Working memory and complex attention processing Ecological Implications Speed of information processing Ecological Implications Recent memory functioning Ecological Implications Visuospatial analysis and visuoconstruction ability Ecological Implications Executive functioning Ecological Implications Mood and psychological functioning Ecological Implications Afterword Acknowledgment References Chapter 32: Neurobehavioral consequences of mild traumatic brain injury in military service members and veterans Introduction TBI in military environments A brief history of brain injury and co-occurring symptoms in war MTBI in the context of polytrauma and co-occurring disorders Differences between OEF/OIF/OND and other war conflicts Overview of the context and case definition Differences between military service-related and civilian MTBI Classification of blast-related injuries Clinical and diagnostic considerations in combat-related MTBI Assessment of service-related TBI Interview as the gold standard for assessment Blast or injury-inducing event Immediate symptoms after TBI in combat theater Postconcussion symptoms Assessment of current cognitive and neurobehavioral symptom complex Co-occurring disorders and MTBI PTSD and MTBI MTBI and other mental health concerns Depression Substance use Sleep problems Self-directed violence Common conditions of polytrauma Assessment of co-occurring disorders Polytrauma rehabilitation and integrated care approaches VHA polytrauma system of care Acute care of concussion Postacute care for MTBI Treatment beyond 6 months Treatment of cognitive sequelae in polytrauma Managing mood after TBI Family support Importance and potential role of peer support and visitation Conclusions Acknowledgment References Chapter 33: Issues in aging following traumatic brain injury Introduction Consequences of aging with a TBI Occupational and social consequences Rehospitalization after TBI Mortality and life expectancy Impact of age-related changes on long-term neurological outcome Tissue loss Cognitive decline Other age-related factors Chronic TBI and neurological disorders Impact of reserve Alzheimer’s disease Epilepsy Other neurological diseases TBI at an advanced age Predictive value of reserve Successful aging References Chapter 34: Children and adolescents: Practical strategies for school participation and transition Introduction Cognitive–communicative challenges after TBI Effect of cognitive–communicative challenges on learning and behaving in the classroom Interrelationship of language, executive functioning, social pragmatics, and self-regulation for a child with cognitive–communicative problems Challenges to language, executive functioning, social communication, and self-regulation for a child with cognitive–communicative problems Challenges to learning after concussion Treatment of cognitive–communicative strengths and needs: An integrative approach for schools Identifying student needs Strategies for addressing underlying cognitive processes Attention/concentration Memory Organization Decreased speed of processing Problem solving Reduced stamina/fatigue Motor deficits Sensory deficits Addressing academic deficits Math Reading Writing Assessing teaching strategies Laws and regulations that affect education, provision of services, and transition for students with TBI IDEA Section 504 Transitioning students with TBI Hospital-to-school transition In-school transitions Postsecondary transition Resources Strategies Summary References Websites for TBI information Websites for concussion information Chapter 35: Long-term discharge planning in traumatic brain injury rehabilitation Introduction Early problem identification during follow-up Avoiding reinjury Activities and activity levels Family systems Caregiver concerns Seizure hygiene Depression Sleep Long-term psychological issues Crisis management Home adaptations Financial planning Additional rehabilitation timing Summary References Appendix 35-A: Family manual outline form Appendix 35-B: Discharge planning checklist Chapter 36: Patients’ rights and responsibilities, health care reform, and telehealth: Ethical considerations Introduction Ethical foundations Patient rights and responsibilities Health care reform: The debate continues Challenges and opportunities of telehealth Conclusions References Index Copyright Title Page Dedication Contents Chapter 1: ‘I’m thinking’ – Oh, but are you? Chapter 2: Renegade perception Chapter 3: The Pushbacker sting Chapter 4: ‘Covid’: The calculated catastrophe Chapter 5: There is no ‘virus’ Chapter 6: Sequence of deceit Chapter 7: War on your mind Chapter 8: ‘Reframing’ insanity Chapter 9: We must have it? So what is it? Chapter 10: Human 2.0 Chapter 11: Who controls the Cult? Chapter 12: Escaping Wetiko Postscript Appendix: Cowan-Kaufman-Morell Statement on Virus Isolation Bibliography Index