Clinical Electrophysiology

Acutely ill patients present with symptoms that don’t immediately yield a diagnosis. Electrophysiological testing can support diagnosis but only if the appropriate tests are ordered.  They must be properly interpreted in conjunction with the actual symptoms. Clinical Electrophysiology presents a wide range of symptoms with specific electrophysiological results. The handbook shows how the complete picture leads to better diagnostic, prognostic or therapeutic conclusions.

The book is organized by the presenting neurological problem in a clinical setting. For each case the authors provide a possible electrophysiological result. This is interpreted and tied to the patient’s symptoms to yield a clinical solution. The handbook avoids theoretical discussion to provide a direct practical guide that:

• Begins with the patient’s symptoms
• Uses a range of electrophysiological modalities
• Shows different test results for similar symptoms
• Relates clinical observation to electrophysiological testing

A final casebook section presents readers with rarer clinical challenges for self-testing.

Providing practical, to-the-point guidance on electrophysiological investigations, Clinical Electrophysiology will guide all neurologists attending acutely ill patients.Content:
Chapter 1 Diffuse and Frontal Fast Activity—Beta (pages 4–5):
Chapter 2 Diffuse Slow Activity–Theta[1–4] (pages 6–7):
Chapter 3 Diffuse Slow Activity—Delta[1–3] (pages 8–10):
Chapter 4 Frontal Intermittent Rhythmic Delta Activity[1–5] (pages 12–13):
Chapter 5 Occipital Intermittent Rhythmic Delta Activity[1–5] (pages 14–15):
Chapter 6 Triphasic Waves[1–7] (pages 16–17):
Chapter 7 Low?Voltage Fast Record without Dominant Alpha Frequencies[1] (pages 18–19):
Chapter 8 Alpha Coma (pages 20–21):
Chapter 9 Spindle Coma[1–5] (pages 22–23):
Chapter 10 Low?Voltage Suppressed Pattern (pages 24–25):
Chapter 11 Burst/Suppression (pages 26–27):
Chapter 12 Diffuse Slowing—Toxic Encephalopathy—Baclofen[1–6] (pages 28–29):
Chapter 13 Diffuse Slowing—Metabolic Encephalopathy—Lithium[1–6] (pages 30–31):
Chapter 14 Diffuse Slowing—Metabolic Encephalopathy—Hypoglycemia[1–3] (pages 32–33):
Chapter 15 Diffuse Slowing—Limbic Encephalopathy[1–6] (pages 34–35):
Chapter 16 Focal Arrhythmic (Polymorphic) Delta Activity (pages 36–37):
Chapter 17 Pseudoperiodic Lateralized Epileptiform Discharges (pages 40–42):
Chapter 18 Bilateral Independent Pseudoperiodic Lateralized Epileptiform Discharges [1–6] (pages 44–45):
Chapter 19 Generalized Periodic Epileptiform Discharges (pages 46–47):
Chapter 20 Frontal Lobe Simple and Complex Partial Seizures[1–5] (pages 52–53):
Chapter 21 Temporal Lobe Simple and Complex Partial Seizures[1–5] (pages 54–55):
Chapter 22 Parietal Lobe Simple Partial Seizures[1–4] (pages 56–57):
Chapter 23 Occipital Lobe Simple Partial Seizures[1–6] (pages 58–59):
Chapter 24 Complex Partial Status Epilepticus—Frontal[6–10] (pages 62–63):
Chapter 25 Complex Partial Status Epilepticus—Temporal[1–4] (pages 64–65):
Chapter 26 Simple Partial Status Epilepticus—Parietal[1–3] (pages 66–67):
Chapter 27 Simple Partial Status Epilepticu—Occipital[1–4] (pages 68–69):
Chapter 28 Generalized Nonconvulsive Status Epilepticus[1–9] (pages 70–72):
Chapter 29 Clinical Definitions of Impaired Responsiveness[1–11] (pages 76–79):
Chapter 30 Locked?In Syndrome—Brainstem Hemorrhage[1–4] (pages 82–83):
Chapter 31 Vegetative State—Postanoxia[1–12] (pages 84–86):
Chapter 32 Minimally Conscious State—After Large, Multifocal Strokes[1–10] (pages 88–89):
Chapter 33 Catatonia—Psychogenic Unresponsiveness/Conversion Disorder[1–5] (pages 90–91):
Chapter 34 Somatosensory Evoked Potential Prognosis in Anoxic Coma[1–8] (pages 92–93):
Chapter 35 Somatosensory Evoked Potential Prognosis in Head Trauma (pages 94–95):
Chapter 36 Somatosensory Evoked Potentials in Midbrain Lesion—Absent Cortical Responses (pages 98–99):
Chapter 37 Somatosensory Evoked Potentials in Diffuse Cortical Anoxic Injury—Absent Cortical and Subcortical Responses[1] (pages 100–101):
Chapter 38 Somatosensory Evoked Potentials in Prolonged Cardiac Arrest—Absence of All Waves above the Brachial Plexus[1,2] (pages 102–103):
Chapter 39 Somatosensory Evoked Potentials after Prolonged Cardiac Arrest—Absence of all Responses Except Cervical N9[1,2] (pages 104–105):
Chapter 40 Somatosensory Evoked Potentials—Median and Tibial after Traumatic Spinal Cord Injury (pages 106–107):
Chapter 41 Visual Evoked Potentials in Worsening Vision (pages 108–109):
Chapter 42 Brainstem Auditory Evoked Potentials—In Worsening Hearing (pages 110–111):
Chapter 43 Causes of Paralysis and Respiratory Failure in the ICU (page 115):
Chapter 44 The Clinical Evaluation of Neuromuscular Disorders (page 116):
Chapter 45 Laboratory Evaluation of Neuromuscular Disorders (page 117):
Chapter 46 Evaluation of Segmental Peripheral Neurological Disorders (page 120):
Chapter 47 Amyotrophic Lateral Sclerosis/Motor Neuropathy (pages 122–123):
Chapter 48 Critical Illness Neuromyopathy (pages 124–126):
Chapter 49 Brachial Plexopathy (pages 128–129):
Chapter 50 Femoral Neuropathy (pages 130–131):
Chapter 51 Sensory Neuropathy/Ganglionopathy[1–3] (pages 132–133):
Chapter 52 Lumbar Radiculopathy[1–3] (pages 134–135):
Chapter 53 Guillain?Barre Syndrome—Demyelinating Polyneuropathy (pages 136–138):
Chapter 54 Myasthenia Gravis—Neuromuscular Junction[1–4] (pages 140–141):
Chapter 55 Myositis—Irritable Myopathy (pages 142–144):
Chapter 56 Statin?Induced Myopathy—Toxic Myopathy/Myalgia (pages 146–148):
Chapter 57 Occipital Blindness and Seizures—Why?[1–4] (pages 149–151):
Chapter 58 Unresponsiveness—Coma, Vegetative State, or Locked?In State? (pages 152–153):
Chapter 59 Unresponsiveness—Organic or Psychogenic?[1,2] (pages 154–155):
Chapter 60 Patient with a Frontal Brain Tumor—Psychiatric Depression, Paranoia, Tumor Growth, or Status Epilepticus?[1–4] (pages 156–157):
Chapter 61 Patient with Idiopathic Generalized Epilepsy on Valproate—Metabolic Encephalopathy or Status Epilepticus?[1–5] (pages 158–159):
Chapter 62 Unresponsiveness—Psychogenic, Encephalopathy, or Limbic Encephalitis?[1–10] (pages 160–161):
Chapter 63 Respiratory Weakness—Toxic or Metabolic? (pages 162–165):
Chapter 64 Failure to Wean from a Ventilator/Internal Ophthalmoplegia—Bulbar Dysfunction, Neuromuscular Junction Problem, or Polyneuropathy? (pages 166–168):
Chapter 65 Progressive Sensory Loss and Painful Gait—Radiculopathy, Toxic or Infectious Neuropathy, or Myopathy? (pages 170–172):
Chapter 66 Slowly Progressive Leg and Arm Weakness—Radiculopathy, Plexopathy, ALS, or CIDP/AMN? (pages 174–176):
Chapter 67 Progressive Thigh Pain and Leg Weakness—Radiculopathy, Vasculitis, Neuropathy, or Amyotrophy? (pages 178–180):